-
Aleyas AG, George JA, Han YW, Rahman MM, Kim SJ, Han SB, Kim BS, Kim K, Eo SK, 2009: Functional modulation of dendritic cells and macrophages by Japanese encephalitis virus through MyD88 adaptor molecule-dependent and -independent pathways[J]. J Immunol, 183, 2462-2474. doi: 10.4049/jimmunol.0801952
-
Bose S, Cho J, 2013: Role of chemokine CCL2 and its receptor CCR2 in neurodegenerative diseases[J]. Arch Pharm Res, 36, 1039-1050. doi: 10.1007/s12272-013-0161-z
-
Campbell GL, Hills SL, Fischer M, Jacobson JA, Hoke CH, Hombach JM, Marfin AA, Solomon T, Tsai TF, Tsu VD, Ginsburg AS, 2011: Estimated global incidence of Japanese encephalitis: a systematic review[J]. Bull World Health Organ, 89, 774A-774E. doi: 10.2471/BLT.10.085233
-
Cao S, Li Y, Ye J, Yang X, Chen L, Liu X, Chen H, 2011: Japanese encephalitis Virus wild strain infection suppresses dendritic cells maturation and function, and causes the expansion of regulatory T cells[J]. Virol J, 8, 39-. doi: 10.1186/1743-422X-8-39
-
Chen ST, Liu RS, Wu MF, Lin YL, Chen SY, Tan DT, Chou TY, Tsai IS, Li L, Hsieh SL, 2012: CLEC5A regulates Japanese encephalitis virus-induced neuroinflammation and lethality[J]. PLoS Pathog, 8, e1002655-. doi: 10.1371/journal.ppat.1002655
-
Chowdhury P, Khan SA (2018) Differential expression levels of inflammatory chemokines and TLRs in patients suffering from mild and severe Japanese encephalitis. Viral Immunol. https://doi.org/10.1089/vim.2018.0103
-
Das S, Ghosh D, Basu A, 2009: Japanese encephalitis virus induce immuno-competency in neural stem/progenitor cells[J]. PLoS ONE, 4, e8134-. doi: 10.1371/journal.pone.0008134
-
Das S, Dutta K, Kumawat KL, Ghoshal A, Adhya D, Basu A, 2011: Abrogated inflammatory response promotes neurogenesis in a murine model of Japanese encephalitis[J]. PLoS ONE, 6, e17225-. doi: 10.1371/journal.pone.0017225
-
Erlanger TE, Weiss S, Keiser J, Utzinger J, Wiedenmayer K, 2009: Past, present, and future of Japanese encephalitis[J]. Emerg Infect Dis, 15, 1-7. doi: 10.3201/eid1501.080311
-
Getts DR, Terry RL, Getts MT, Muller M, Rana S, Shrestha B, Radford J, Van Rooijen N, Campbell IL, King NJ, 2008: Ly6c+ "inflammatory monocytes" are microglial precursors recruited in a pathogenic manner in West Nile virus encephalitis[J]. J Exp Med, 205, 2319-2337. doi: 10.1084/jem.20080421
-
Glass WG, Lim JK, Cholera R, Pletnev AG, Gao JL, Murphy PM, 2005: Chemokine receptor CCR11 promotes leukocyte trafficking to the brain and survival in West Nile virus infection[J]. J Exp Med, 202, 1087-1098. doi: 10.1084/jem.20042530
-
Gouwy M, Struyf S, Catusse J, Proost P, Van Damme J, 2004: Synergy between proinflammatory ligands of G protein-coupled receptors in neutrophil activation and migration[J]. J Leukoc Biol, 76, 185-194. doi: 10.1189/jlb.1003479
-
Gupta N, Rao PV, 2011: Transcriptomic profile of host response in Japanese encephalitis virus infection[J]. Virol J, 8, 92-. doi: 10.1186/1743-422X-8-92
-
Han YW, Choi JY, Uyangaa E, Kim SB, Kim JH, Kim BS, Kim K, Eo SK, 2014: Distinct dictation of Japanese encephalitis virus-induced neuroinflammation and lethality via triggering TLR3 and TLR4 signal pathways[J]. PLoS Pathog, 10, e1004319-. doi: 10.1371/journal.ppat.1004319
-
Hosking MP, Lane TE, 2010: The role of chemokines during viral infection of the CNS[J]. PLoS Pathog, 6, e1000937-. doi: 10.1371/journal.ppat.1000937
-
Ireland DD, Tami C, Pedras-Vasconcelos J, Verthelyi D, 2017: CD4 and CD8 T cells mediate distinct lethal meningoencephalitis in mice challenged with Tacaribe arenavirus[J]. Cell Mol Immunol, 14, 90-107. doi: 10.1038/cmi.2016.41
-
Jain N, Oswal N, Chawla AS, Agrawal T, Biswas M, Vrati S, Rath S, George A, Bal V, Medigeshi GR, 2017: CD8 T cells protect adult naive mice from JEV-induced morbidity via lytic function[J]. PLoS Negl Trop Dis, 11, e0005329-. doi: 10.1371/journal.pntd.0005329
-
Jin HT, Anderson AC, Tan WG, West EE, Ha SJ, Araki K, Freeman GJ, Kuchroo VK, Ahmed R, 2010: Cooperation of Tim-3 and PD-1 in CD8 T-cell exhaustion during chronic viral infection[J]. Proc Natl Acad Sci U S A, 107, 14733-14738. doi: 10.1073/pnas.1009731107
-
Kaushik DK, Gupta M, Kumawat KL, Basu A, 2012: NLRP3 inflammasome: key mediator of neuroinflammation in murine Japanese encephalitis[J]. PLoS ONE, 7, e32270-. doi: 10.1371/journal.pone.0032270
-
Kim HJ, Verbinnen B, Tang X, Lu L, Cantor H, 2010: Inhibition of follicular T-helper cells by CD8(+) regulatory T cells is essential for self tolerance[J]. Nature, 467, 328-332. doi: 10.1038/nature09370
-
Kim JH, Patil AM, Choi JY, Kim SB, Uyangaa E, Hossain FM, Park SY, Lee JH, Kim K, Eo SK, 2016: CCL2, but not its receptor, is essential to restrict immune privileged central nervous system-invasion of Japanese encephalitis virus via regulating accumulation of CD11b(+) Ly-6C(hi) monocytes[J]. Immunology, 149, 186-203. doi: 10.1111/imm.12626
-
Lannes N, Neuhaus V, Scolari B, Kharoubi-Hess S, Walch M, Summerfield A, Filgueira L, 2017: Interactions of human microglia cells with Japanese encephalitis virus[J]. Virol J, 14, 8-. doi: 10.1186/s12985-016-0675-3
-
Larena M, Regner M, Lobigs M, 2012: The chemokine receptor CCR23, a therapeutic target for HIV/AIDS antagonists, is critical for recovery in a mouse model of Japanese encephalitis[J]. PLoS ONE, 7, e44834-. doi: 10.1371/journal.pone.0044834
-
Li F, Wang Y, Yu L, Cao S, Wang K, Yuan J, Wang C, Wang K, Cui M, Fu ZF, 2015: viral infection of the central nervous system and neuroinflammation precede blood-brain barrier disruption during Japanese encephalitis virus infection[J]. J Virol, 89, 5602-5614. doi: 10.1128/JVI.00143-15
-
Lim JK, Obara CJ, Rivollier A, Pletnev AG, Kelsall BL, Murphy PM, 2011: Chemokine receptor Ccr2 is critical for monocyte accumulation and survival in West Nile virus encephalitis[J]. J Immunol, 186, 471-478. doi: 10.4049/jimmunol.1003003
-
Lv BM, Tong XY, Quan Y, Liu MY, Zhang QY, Song YF, Zhang HY (2018) Drug repurposing for Japanese encephalitis virus infection by systems biology methods. Molecules.https://doi.org/10.3390/molecules23123346
-
Michlmayr D, Lim JK, 2014: Chemokine receptors as important regulators of pathogenesis during arboviral encephalitis[J]. Front Cell Neurosci, 8, 264-.
-
Nansen A, Marker O, Bartholdy C, Thomsen AR, 2000: CCR28+ and CCR28+CD8+ T cells increase during viral infection and migrate to sites of infection[J]. Eur J Immunol, 30, 1797-1806. doi: 10.1002/1521-4141(200007)30:7<1797::AID-IMMU1797>3.0.CO;2-B
-
Ransohoff RM, Engelhardt B, 2012: The anatomical and cellular basis of immune surveillance in the central nervous system[J]. Nat Rev Immunol, 12, 623-635. doi: 10.1038/nri3265
-
Sharpe AH, Wherry EJ, Ahmed R, Freeman GJ, 2007: The function of programmed cell death 1 and its ligands in regulating autoimmunity and infection[J]. Nat Immunol, 8, 239-245. doi: 10.1038/ni1443
-
Singh A, Kulshreshtha R, Mathur A, 2000: Secretion of the chemokine interleukin-8 during Japanese encephalitis virus infection[J]. J Med Microbiol, 49, 607-612. doi: 10.1099/0022-1317-49-7-607
-
Solomon T, Dung NM, Kneen R, Gainsborough M, Vaughn DW, Khanh VT, 2000: Japanese encephalitis[J]. J Neurol Neurosurg Psychiatry, 68, 405-415. doi: 10.1136/jnnp.68.4.405
-
Tiwari S, Singh RK, Tiwari R, Dhole TN, 2012: Japanese encephalitis: a review of the Indian perspective[J]. Braz J Infect Dis, 16, 564-573. doi: 10.1016/j.bjid.2012.10.004
-
Wang H, Liang G, 2015: Epidemiology of Japanese encephalitis: past, present, and future prospects[J]. Ther Clin Risk Manag, 11, 435-448. doi: 10.2147/TCRM.S51168
-
Winter PM, Dung NM, Loan HT, Kneen R, Wills B, le Thu T, House D, White NJ, Farrar JJ, Hart CA, Solomon T, 2004: Proinflammatory cytokines and chemokines in humans with Japanese encephalitis[J]. J Infect Dis, 190, 1618-1626. doi: 10.1086/423328
-
Yang Y, Ye J, Yang X, Jiang R, Chen H, Cao S, 2011: Japanese encephalitis virus infection induces changes of mRNA profile of mouse spleen and brain[J]. Virol J, 8, 80-. doi: 10.1186/1743-422X-8-80
-
Yurchenko E, Tritt M, Hay V, Shevach EM, Belkaid Y, Piccirillo CA, 2006: CCR37-dependent homing of naturally occurring CD4+ regulatory T cells to sites of Leishmania major infection favors pathogen persistence[J]. J Exp Med, 203, 2451-2460. doi: 10.1084/jem.20060956
-
Zelinskyy G, Myers L, Dietze KK, Gibbert K, Roggendorf M, Liu J, Lu M, Kraft AR, Teichgraber V, Hasenkrug KJ, Dittmer U, 2011: Virus-specific CD8+ T cells upregulate programmed death-1 expression during acute friend retrovirus infection but are highly cytotoxic and control virus replication[J]. J Immunol, 187, 3730-3737. doi: 10.4049/jimmunol.1101612