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Volume 28 Issue 4
August 2013

    Muaz M. Abdellatif, Bashir Salim, Awad A. Ibrahim, Tigani Asil and Abdelmalik I. Khalafalla. Analysis of TK and C18L Genes of Wild-type and Cell Culture Passaged Camelpox Virus[J]. Virologica Sinica, 2013, 28(4): 239-241. doi: 10.1007/s12250-013-3329-2
    Citation: Muaz M. Abdellatif, Bashir Salim, Awad A. Ibrahim, Tigani Asil, Abdelmalik I. Khalafalla. Analysis of TK and C18L Genes of Wild-type and Cell Culture Passaged Camelpox Virus .VIROLOGICA SINICA, 2013, 28(4) : 239-241.  http://dx.doi.org/10.1007/s12250-013-3329-2
    shu

    Analysis of TK and C18L Genes of Wild-type and Cell Culture Passaged Camelpox Virus

    cstr: 32224.14.s12250-013-3329-2
    • 1.

      Department of Microbiology, Faculty of Veterinary Medicine, University of Khartoum, Shambat 13314, Sudan;

    • 2.

      Department of Microbiology, Faculty of Veterinary Science, University of Nyala, Southern Darfur, P.OBox 155 Nyala, Sudan;

    • 3.

      Department of Pathology, Faculty of Veterinary Science, University of Nyala, Southern Darfur, P.OBox 155 Nyala, Sudan

    • 通讯作者： Abdelmalik I. Khalafalla, abdokhlf@yahoo.co.uk
    • 收稿日期： 2013-05-15
      录用日期： 2013-07-09
      出版日期： 2013-07-24

    • Camelpox is an infectious skin disease of camels caused by camelpox virus (CMLV). It is confined to camel rearing belts of developing countries (Wernery U, et al., 2002). CMLV is classified in the Orthopoxvirus (OPV) genus of the subfamily Chordopoxvirinae in the Poxviridae family.

    Analysis of TK and C18L Genes of Wild-type and Cell Culture Passaged Camelpox Virus

    • 1.

      Department of Microbiology, Faculty of Veterinary Medicine, University of Khartoum, Shambat 13314, Sudan

    • 2.

      Department of Microbiology, Faculty of Veterinary Science, University of Nyala, Southern Darfur, P.O. Box 155 Nyala, Sudan

    • 3.

      Department of Pathology, Faculty of Veterinary Science, University of Nyala, Southern Darfur, P.O. Box 155 Nyala, Sudan

    • Corresponding author: Abdelmalik I. Khalafalla, abdokhlf@yahoo.co.uk
    • Received Date: 15 May 2013
      Accepted Date: 09 July 2013
      Published Date: 24 July 2013
    • Camelpox is an infectious skin disease of camels caused by camelpox virus (CMLV). It is confined to camel rearing belts of developing countries (Wernery U, et al., 2002). CMLV is classified in the Orthopoxvirus (OPV) genus of the subfamily Chordopoxvirinae in the Poxviridae family.
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      1. Balamurugan V, Hosamani M, Jayappa K D, Venkatesan B, Chauhan R, Singh A. 2009. A polymerase chain reaction strategy for the diagnosis of camelpox.Vet Diag Invest, 21:231-237.
          doi: 10.1177/104063870902100209

      2. Binns M, Mumford J, Wernery W. 1992. Analysis of the Camelpox virus thymidine kinase gene. Br Vet J, 148:541-546.
          doi: 10.1016/0007-1935(92)90010-X

      3. Dietzschold B, Wunner W H, Wiktor T J, Lopes A D, Lafon M, Smith C L, Koprowski H. 1983. Characterization of an antigenic determinant of the glycoprotein that correlates with pathogenicity of rabies virus. Proc Natl Acad Sci. USA, 80:70-74.
          doi: 10.1073/pnas.80.1.70

      4. Duraffour S, Meyer H, Andrei G, Snoeck R. 2011. Camelpox virus. Antiviral Res, 92(2):167-86.
          doi: 10.1016/j.antiviral.2011.09.003

      5. Hafez S M, Al-Sukayran A, Dela Cruz D M, Mazloum K S, Al-Bokmy A M, Al-Mukayel A, Amjad A M. 1992. Development of a live cell culture camelpox vaccine. Vaccine, 10:533-537.
          doi: 10.1016/0264-410X(92)90353-L

      6. Khalafalla A I, Mohamed M E M. 1996. Clinical and epizootiological features of camelpox in eastern Sudan. J Camel Pract and Res, 2:99-102.

      7. Khalafalla A I, Mohamed M E H, Ali B H. 1998. Camel pox in the Sudan. 1-isolation and identification of the causative virus. J Camel Pract and Res, 5:229-233.

      8. Laidlaw S M, Skinner M A. 2004. Comparison of the genome sequence of FP9, an attenuated, tissue culture-adapted European strain of fowlpox virus, with those of virulent American and European viruses. J Gen Virol, 85:305-322.
          doi: 10.1099/vir.0.19568-0

      9. Massung R F, Esposito J J, Liu L Q, Utterback T R, Knight J C, Aubin L, Yuran T E, Parsons J M, Loparev V N, Selivanov N A, Cavallaro K F, Kerlavage A R, Mahy B W, Venter J C. 1993. Potential virulence determinants in terminal regions of variola smallpox virus genome. Nature, 366:748-751.
          doi: 10.1038/366748a0

      10. Monath T P. 2005.Yellow fever vaccine. Expert Rev. Vaccines, 4:553-574.
          doi: 10.1586/14760584.4.4.553

      11. Nguyen Ba-Vy, Guerre L, Martin-Sant G. 1996. Etude preliminaire de I innocuite e du pouvoir immunogene de la ouche attenuee VD47/25 de camelpox virus. Revue de Elev Med Vet Pays trop, 49:189-194.

      12. Perkus M E, Goebel S J, Davis S W, Johnson G P, Limbach K, Norton E K, Paoletti E. 1991. Deletion of 55 open reading frames from the termini for vaccinia virus. Virology, 180:406-410.
          doi: 10.1016/0042-6822(91)90047-F

      13. Pfeffer M, Meyer H, Wernery U, Kaaden O R. 1996. Comparison of camelpox viruses isolated in Dubai. Vet Microbiol, 49:135-146.
          doi: 10.1016/0378-1135(95)00181-6

      14. Singh R K, Balamurugan V, Hosamani M, Kallesh D J, Bhanuprakash V. 2008. Sequence analysis of C18L gene of buffalopox virus:PCR strategy for specific detection and differentiation of buffalopox from orthopoxviruses. J Virol Methods, 154(1-2):146-153.
          doi: 10.1016/j.jviromet.2008.08.009

      15. Tulman E R, Afonso C L, Lu Z, Zsak L, Sur J H, Sandybaev N T, Karembekova U Z, Zaitsev V L, Kutish G F, Rock D L. 2002. The genomes of sheeppox and goatpox viruses. J Virol, 76:6054-6061.
          doi: 10.1128/JVI.76.12.6054-6061.2002

      16. Westrop G D, Wareham K A, Evans D M, Dunn G, Minor P D, Magrath D I, Taffs F, Marsden S, Skinner M A, Schild G C, Almond J W. 1989. Genetic basis of attenuation of the Sabin type 3 oral poliovirus vaccine. J Virol, 63:1338-1344.

      17. Wernery U, Kaaden O R. 2002. Camel pox. In: Infectious Diseases in Camelids, Second Edition, Wernery U, Kaaden O R, eds, Blackwell Science Berlin, Vienna: 176-185.

      18. Wernery U, Zachariah R. 1999. Experimental camelpox infection in vaccinated and unvaccinated dromedaries. J Vet Med (B) Infect Dis, 46:131-136.

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      Analysis of TK and C18L Genes of Wild-type and Cell Culture Passaged Camelpox Virus

        Corresponding author: Abdelmalik I. Khalafalla, abdokhlf@yahoo.co.uk
      • 1. Department of Microbiology, Faculty of Veterinary Medicine, University of Khartoum, Shambat 13314, Sudan
      • 2. Department of Microbiology, Faculty of Veterinary Science, University of Nyala, Southern Darfur, P.O. Box 155 Nyala, Sudan
      • 3. Department of Pathology, Faculty of Veterinary Science, University of Nyala, Southern Darfur, P.O. Box 155 Nyala, Sudan
      • Received Date: 15 May 2013
      • Accepted Date: 09 July 2013
      • Published Date: 24 July 2013

      Abstract: Camelpox is an infectious skin disease of camels caused by camelpox virus (CMLV). It is confined to camel rearing belts of developing countries (Wernery U, et al., 2002). CMLV is classified in the Orthopoxvirus (OPV) genus of the subfamily Chordopoxvirinae in the Poxviridae family.

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